ArticleCureus2026
Breast Metastasis From Cervical Small Cell Neuroendocrine Carcinoma Mimicking Primary Breast Cancer.
Article in Cureus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
0 citing papers in PubMed.
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
We describe a case of breast metastasis originating from a small-cell neuroendocrine carcinoma (SCNEC) of the uterine cervix, an uncommon presentation that can be difficult to distinguish from a primary breast malignancy. We report a 29-year-old woman treated three years earlier for FIGO stage IB2 cervical SCNEC with chemotherapy, surgery, and radiotherapy, who presented with a 1.5-cm mass in the left breast. Imaging suggested a primary breast malignancy, but her oncologic history raised concern for metastasis. Core-needle biopsy showed a high-grade small-cell neoplasm expressing synaptophysin, chromogranin A, and CD56, with a high Ki-67 index (>80%); breast-lineage markers, including GATA3 and mammaglobin, were negative. Whole-body fluorodeoxyglucose positron emission tomography-computed tomography (FDG PET-CT) showed an isolated hypermetabolic breast lesion without other disease. Nipple-sparing mastectomy with immediate reconstruction was performed, followed by platinum-etoposide chemotherapy; at 18 months, there was no evidence of recurrence. Breast metastasis from cervical SCNEC can mimic primary breast cancer; diagnosis relies on integrating clinical history, imaging, and immunohistochemistry. Selected patients with isolated disease may benefit from individualised multimodal therapy.
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