ReviewInternational journal of general medicine2026
Mechanisms by Which the Cervicovaginal Microbiota Mediates Endometrial Repair Following Uterine Injury: A Review of Recent Advances.
Review in International journal of general medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
7 authors.
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Abstract
Aim: To summarize current evidence on the molecular mechanisms by which the cervicovaginal microbiota mediates post-injury endometrial repair and to discuss relevant microbiota-targeted intervention strategies. Methods: A narrative review was conducted based on relevant English-language literature retrieved from PubMed, Embase, and Web of Science up to April 30, 2026. The literature was synthesized with a focus on the role of the cervicovaginal microbiota in endometrial repair through the TGF-β1/Smad, TLR4/NF-κB, and Wnt/β-catenin signaling pathways, as well as relevant microbiota-targeted intervention strategies. Results: Cervicovaginal microbiota dysbiosis promotes endometrial fibrosis and impairs tissue regeneration via dysregulation of the above signaling pathways. Conclusion: The cervicovaginal microbiota is an important regulator of post-injury endometrial repair. Microbiota-targeted interventions represent a promising therapeutic direction, though current evidence remains largely observational and further high-quality clinical trials are needed.
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