Evidence map›Paper›PMID 42795322›Full record

ArticleLife (Basel, Switzerland)2026

The Preservation of Functional Reserve: A Control Systems Framework for Human Aging.

Robert T O'Leary

Abstract read
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In one paragraph

Article in Life (Basel, Switzerland), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Robert T O'LearyOrlando College of Osteopathic Medicine, Orlando, FL 34787, USA.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The dominant clinical manifestation of aging is not mortality but the progressive depletion of functional reserve-the surplus physiological capacity separating independent function from disability. Existing geroscience frameworks describe molecular aging in detail yet give the point-of-care clinician little basis for prioritizing targets. We reframe functional aging as a control systems problem in which aging is modeled as progressive instability within a coupled, bidirectional mitochondrial-epigenetic axis. That axis is called central in two limited senses only: network connectivity and therapeutic tractability; no causal primacy is claimed over mTOR signaling, inflammaging, senescence, proteostasis failure, telomere attrition, or stem cell exhaustion. Candidate capacities were scored against three conjunctive criteria-independence, necessity, and modifiability. Five satisfy them at the thresholds stated, and sensitivity analysis reports which threshold changes would instead yield four or seven, so the count follows from a stated procedure rather than standing as a natural constant. Interventions are mapped onto these capacities in a separate layer. The result is a hierarchically ordered, clinically actionable, falsifiable architecture in which functional reserve is the primary therapeutic target and lifespan a downstream consequence-and whose primary test, deletion of any single capacity from longitudinal models, is evaluable in existing cohorts without new data collection.

Indexed as

control systemsfunctional reservehomeostenosislongevity medicinemitochondrial–epigenetic axisphysiologic resilience

Identifiers

PMID42795322

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.