Evidence map›Paper›PMID 42794713›Full record

ArticleInternational journal of molecular sciences2026

Physiological and Proteomic Analyses Reveal the Adaptive Mechanisms of Maize Genotypes Under Potassium Deficiency.

Xudong Yang, Haoran Qin, Fei Zou, Hui Ma, Hui Li, Yanye Ruan, Shuisen Chen

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

7 authors.

Xudong YangCollege of Bioscience and Biotechnology, Shenyang Agricultural University, Shenyang 110866, China.
Haoran QinCollege of Bioscience and Biotechnology, Shenyang Agricultural University, Shenyang 110866, China.
Fei ZouMaize Research Institute, Hulunbuir Academy of Agricultural and Animal Husbandry Sciences, Zhalantun 162650, China.
Hui MaCollege of Bioscience and Biotechnology, Shenyang Agricultural University, Shenyang 110866, China.
Hui LiCollege of Bioscience and Biotechnology, Shenyang Agricultural University, Shenyang 110866, China.
Yanye RuanCollege of Bioscience and Biotechnology, Shenyang Agricultural University, Shenyang 110866, China.
Shuisen ChenCollege of Bioscience and Biotechnology, Shenyang Agricultural University, Shenyang 110866, China.ORCID 0000-0001-7932-163X

Funding

National Natural Science Foundation of China 31601808
6 · The paper itself

Abstract

Potassium (K) deficiency severely constrains maize productivity, yet the molecular mechanisms underlying genotypic differences in K tolerance remain poorly understood. A comparative physiological and iTRAQ-based quantitative proteomic analysis was conducted on two maize inbred lines with contrasting K-deficiency tolerance (Ktm, tolerant; Ksm, sensitive) following 3 days of K starvation (0 mM K), using roots as the primary analytical target. Physiological assessments revealed that under K deficiency, Ktm exhibited higher root vitality (Ktm decreased by 16.65% vs. Ksm by 31.54%) and larger root volume but lower electrolyte leakage compared with Ksm. Proteomic profiling identified 93 and 126 differentially abundant proteins (DAPs) in Ktm and Ksm, respectively. Integrative analysis indicated that Ktm responsed to K deprivation though coordinated downregulation of glycolytic enzymes, differential ROS accumulation with enhanced catalase (CAT) and peroxidase (POD) upregulation, and increased the abundances of cell wall-reinforcing proteins (dirigent proteins and cinnamyl alcohol dehydrogenase). In contrast, Ksm exhibited a less efficient stress response characterized by impaired ROS signaling, H

Indexed as

Adaptation, PhysiologicalPlant ProteinsPotassiumPotassium DeficiencyProteomeProteomicsZea maysGene Expression Regulation, PlantGenotypePlant RootsReactive Oxygen SpeciesPlant ProteinsPotassiumProteomeReactive Oxygen SpeciesiTRAQmaizepotassiumproteomicsroots

Identifiers

PMID42794713
PMCPMC13607648

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.