ArticleEnvironment & health (Washington, D.C.)2026
Gestational Low-Dose Bisphenol A Exposure Enhances Susceptibility to Metabolic Disorders in Adult Offspring Mice: Role of the Gut Microbiota-SCFAs-GPRs Axis.
Article in Environment & health (Washington, D.C.), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Abstract
Bisphenol A (BPA), frequently detected at low levels in pregnant women, has been linked to long-term metabolic disturbances in offspring. Emerging evidence indicates that maternal gut microbiota critically influence offspring susceptibility to metabolic diseases through their metabolites. This study aimed to investigate how maternal gut microbiota and short-chain fatty acids (SCFAs) mediate the increased susceptibility to high-fat-diet (HFD)-induced metabolic disorders in adult offspring following gestational low-dose BPA exposure. Pregnant mice were exposed to 0, 10, 100, and 1000 nmol/L BPA via drinking water throughout gestation, after which pups were cross-fostered by unexposed dams and fed normal chow or HFD until adulthood. Adult offspring from BPA-exposed dams exhibited aggravated HFD-induced dyslipidemia and insulin resistance. Maternal BPA exposure decreased the abundance of f_
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