ArticlePlant physiology2026
A peach esterase/lipase mediates lipid remodeling as a protective mechanism against chilling injury.
Article in Plant physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
11 authors.
Funding
Abstract
Peach fruit is highly susceptible to chilling injury, which induces internal browning during long-term low-temperature storage. Internal browning was found to occur only at intermediate temperatures (5, 8, and 12 °C), but not at either lower (0 °C) or higher (16 °C) temperatures. Proteomic analysis of peach fruit stored at different temperatures for various durations was performed, and a GDSL esterase/lipase, PpGELP18, a protein with in vitro esterase/lipase activity, was identified. Transgenic tobacco (Nicotiana tabacum) overexpressing PpGELP18 demonstrated enhanced chilling tolerance. Lipidomic analysis of transgenic tobacco (N. tabacum) leaves and peach fruit revealed that PpGELP18 is involved in intracellular lipid remodeling, particularly in the accumulation of membrane phospholipids. Elevated levels of unsaturated phosphatidylcholines and phosphatidylethanolamines, along with a decrease in lysophospholipids and diacylglycerols, were observed in transgenic tobacco (N. tabacum). In contrast, a decrease in phospholipids and an increase in diacylglycerols occurred in peach fruit at chilling-injury-inducing temperatures and during the late storage period at 5 °C, accompanied by internal browning. Ultrastructural examination confirmed that chilling-injury-associated internal browning was accompanied by damage to the plasma membrane and tonoplast. These results demonstrate that elevated PpGELP18 confers chilling tolerance in both peach fruit and tobacco (N. tabacum) plants by modulating lipid composition.
Indexed as
Identifiers
42764161What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.