Evidence map›Paper›PMID 42751788›Full record

ReviewArteriosclerosis, thrombosis, and vascular biology2026

Pulmonary Alveolar Proteinosis: Rethinking Surfactant Homeostasis Through Alveolar Macrophage Lipid-Handling Dysfunction.

Elinor Lee, Isaiah J Little, Lucas Dantas, Elizabeth J Tarling

Abstract readReview
In one paragraph

Review in Arteriosclerosis, thrombosis, and vascular biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Elinor LeeDivision of Pulmonary, Critical Care, and Sleep Medicine, Department of Medicine (E.L.), University of California, Los Angeles.ORCID 0000-0003-3008-8325
Isaiah J LittleMolecular Biology Institute (I.J.L., E.J.T.), University of California, Los Angeles.ORCID 0000-0002-3073-553X
Lucas DantasSchool of Medicine, University College Dublin, Ireland (L.D.).
Elizabeth J TarlingMolecular Biology Institute (I.J.L., E.J.T.), University of California, Los Angeles.ORCID 0000-0002-0599-0432

Funding

Targeting the gut-liver axis in cardiovascular diseaseR01HL163908 · NHLBI · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI Elizabeth Joanna Tarling, Thomas A Vallim · 2023 to 2026
$2.9M
Post-transcriptional regulation of LDLRR01HL174008 · NHLBI · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI Elizabeth Joanna Tarling, Thomas A Vallim · 2024 to 2026
$2.5M
Post-transcriptional regulation of hepatokines and systemic metabolismR01DK144132 · NIDDK · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI Elizabeth Joanna Tarling, Thomas A Vallim · 2025 to 2026
$1.6M
Role of GM-CSF and impaired lipid metabolism in the pathogenesis of PAPK08HL166970 · NHLBI · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI Elinor Lee · 2024 to 2026
$512k
NHLBI NIH HHS K08 HL166970NHLBI NIH HHS R01 HL163908NHLBI NIH HHS R01 HL174008NIDDK NIH HHS R01 DK144132
6 · The paper itself

Abstract

Pulmonary alveolar proteinosis is a rare lung disease characterized by the accumulation of surfactant lipids and proteins within the alveolar space, leading to impaired gas exchange and progressive respiratory dysfunction. In most cases, pulmonary alveolar proteinosis arises from disruption of GM-CSF (granulocyte-macrophage colony-stimulating factor) signaling, resulting in defective alveolar macrophage maturation and a failure of surfactant clearance. Emerging work has reframed pulmonary alveolar proteinosis as a disorder of macrophage lipid handling, linking impaired cholesterol efflux and surfactant catabolism to both disease initiation and progression. In this review, we synthesize current understanding of pulmonary alveolar proteinosis pathophysiology, highlighting GM-CSF-dependent regulation of alveolar macrophage function, insights from animal and cellular models, and the evolving therapeutic landscape. We further discuss how targeting macrophage lipid homeostasis may offer new disease-modifying strategies.

Indexed as

Lipid MetabolismMacrophages, AlveolarPulmonary Alveolar ProteinosisPulmonary Surfactant-Associated ProteinsPulmonary SurfactantsAnimalsCholesterolGranulocyte-Macrophage Colony-Stimulating FactorHomeostasisHumansSignal TransductionCholesterolGranulocyte-Macrophage Colony-Stimulating FactorPulmonary Surfactant-Associated ProteinsPulmonary Surfactantsbiomarkerscholesterolhomeostasislipidsmacrophage

Identifiers

PMID42751788
PMCPMC13686622

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.