ArticleJournal of molecular and cellular cardiology plus2026
Netrin-1 suppresses CCL2-driven macrophage migration: implications for modulating vascular inflammation and aortic sinus enlargement in hyperlipidaemic mice.
Article in Journal of molecular and cellular cardiology plus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Background: Atherosclerosis is driven by sustained recruitment and accumulation of monocytes and macrophages within arterial walls through chemokine-mediated pathways. Netrin-1, an immunomodulatory molecule originally identified in neural development, has emerged as a potential regulator of vascular inflammation, though its precise effects on chemokine-driven leukocyte migration remain undefined. Methods: We investigated netrin-1 effects on macrophage migration Results: Netrin-1 selectively inhibited CCL2-driven macrophage migration Conclusions: Our data suggests that systemic netrin-1 selectively inhibits chemokine-driven monocyte migration
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