Evidence map›Paper›PMID 42729564›Full record

ArticleClinical case reports2026

TTP-Like Syndrome and Subsequent Non-Aneurysmal Subarachnoid Hemorrhage in HbSC Disease: A Case Report.

Benjamin Vieten, Jan Vorwerk, Jan Morf, Alice Dauth, Stefan Sondermann, Peter Schramm, Tobias Graf, Nikolas von Bubnoff, Lorenz Oelschläger, Cyrus Khandanpour

Abstract read
In one paragraph

Article in Clinical case reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Benjamin VietenDepartment of Hematology and Oncology, University Cancer Center Schleswig-Holstein (UCCSH), University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0009-0004-0538-3466
Jan VorwerkDepartment of Hematology and Oncology, University Cancer Center Schleswig-Holstein (UCCSH), University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0000-0002-0568-8970
Jan MorfDepartment of Hematology and Oncology, University Cancer Center Schleswig-Holstein (UCCSH), University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.
Alice DauthDepartment of Neurosurgery, University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0000-0002-9329-1550
Stefan SondermannDepartment of Neuroradiology, University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0000-0002-6973-0824
Peter SchrammDepartment of Neuroradiology, University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.
Tobias GrafDepartment of Cardiology, Angiology and Intensive Care Medicine, University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0000-0003-4038-147X
Nikolas von BubnoffDepartment of Hematology and Oncology, University Cancer Center Schleswig-Holstein (UCCSH), University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0000-0001-9593-8947
Lorenz OelschlägerDepartment of Hematology and Oncology, University Cancer Center Schleswig-Holstein (UCCSH), University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0009-0002-8960-5040
Cyrus KhandanpourDepartment of Hematology and Oncology, University Cancer Center Schleswig-Holstein (UCCSH), University Hospital Schleswig-Holstein (UKSH) University of Luebeck Luebeck Germany.ORCID https://orcid.org/0000-0003-4655-6269

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Sickle cell disease (SCD) with hemoglobin- (Hb-) SC genotype is often considered a milder SCD variant, yet life-threatening complications can occur. A 26-year-old man with HbSC disease presented with an infection triggered vaso-occlusive crisis (VOC), acute chest syndrome (ACS), severe thrombocytopenia, Coombs-negative hemolysis with schistocytes, acute kidney injury, and markedly elevated lactate dehydrogenase (LDH) in the setting of

Indexed as

fat embolism syndromeHbSCsickle cell diseasesubarachnoid hemorrhageTTPTTP‐like syndrome

Identifiers

PMID42729564
PMCPMC13563050

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.