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ArticleHypertension research : official journal of the Japanese Society of Hypertension2026

Adipocyte-specific deletion of adrenomedullin exacerbates high-fat diet-induced hypertension.

Miku Oya, Kosai Cho, Hideaki Inazumi, Toshio Nishikimi, Koichiro Kuwahara, Naoto Minamino, Yasuaki Nakagawa, Hideyuki Kinoshita, Kenji Moriuchi, Hiromu Yanagisawa and 6 more

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Article in Hypertension research : official journal of the Japanese Society of Hypertension, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

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No citing paper in PubMed yet.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

16 authors.

Miku Oya *Department of Cardiovascular Medicine, Shinshu University School of Medicine, Matsumoto, Japan.
Kosai Cho *Department of Emergency and Critical Care Medicine, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.
Hideaki Inazumi *Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan. hinazumi@kuhp.kyoto-u.ac.jp.
Toshio NishikimiDepartment of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan. nishikim@kuhp.kyoto-u.ac.jp.
Koichiro KuwaharaDepartment of Cardiovascular Medicine, Shinshu University School of Medicine, Matsumoto, Japan. kkuwah@shinshu-u.ac.jp.
Naoto MinaminoNational Cerebral and Cardiovascular Center Research Institute, Suita, Osaka, Japan.
Yasuaki NakagawaDepartment of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan.
Hideyuki KinoshitaDepartment of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan.
Kenji MoriuchiDepartment of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan.
Hiromu YanagisawaDepartment of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan.
Takahiko KanamoriDepartment of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan.
Takeya MinamiMedical Innovation Center, Kyoto University Graduate School of Medicine, Kyoto, Japan.
Eiichiro NishiDepartment of Pharmacology, Shiga University of Medical Science, Otsu, Japan.
Kenji KangawaNational Cerebral and Cardiovascular Center Research Institute, Suita, Osaka, Japan.
Kazuwa NakaoMedical Innovation Center, Kyoto University Graduate School of Medicine, Kyoto, Japan.
Koh OnoDepartment of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Sakyo-ku, Kyoto, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Adrenomedullin (AM) is highly expressed in cardiovascular tissues and has been extensively studied as a vasoactive peptide. Subsequent studies have also demonstrated substantial AM expression in adipose tissue; however, the pathophysiological role of adipocyte-derived AM in obesity remains unclear. We generated adipocyte-specific AM knockout mice (Adiponectin-Cre; Adm^fl/fl, cKO) and fed cKO and littermate controls normal chow (NC) or HFD from 8 to 36 weeks of age. HFD increased systolic blood pressure (SBP) in both genotypes, but SBP was higher in cKO mice under chronic HFD conditions, despite no genotype-dependent difference in body weight. Circulating AM increased with HFD in both groups but remained lower in cKO mice. Plasma renin concentration did not differ by diet or genotype, whereas aldosterone increased with HFD without significant genotype-dependent differences. Urinary epinephrine and norepinephrine were unchanged, whereas an HFD-associated increase in urinary dopamine observed in controls was attenuated in cKO mice. Together, these data suggest that adipocyte-derived AM mitigates HFD-associated hypertension and may be linked to renal dopaminergic responses during chronic metabolic stress.

Indexed as

AdipocyteAdrenomedullinDopamineHypertensionObesity

Identifiers

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.