ArticleFrontiers in plant science2026
Day/night temperature fluctuations delay the high-temperature-induced decline in Cry1Ac endotoxin concentration in cotton squares.
Article in Frontiers in plant science, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Introduction: High-temperature stress significantly impacts Cry1Ac endotoxin efficacy in Methods: Hence, a 2-year (2021-2022) plot experiment was carried out to investigate the response of square Cry1Ac endotoxin concentration to fluctuating high temperature (HDT/NNT, 38/27 °C) compared with a control treatment (CK, 32/25 °C) in cultivars 'Sikang 1' and 'Sikang 3'. Results: The results indicated that the Cry1Ac endotoxin concentration remained stable under CK but decreased significantly under HDT/NNT only after 7 days of stress, with the decline intensifying over time. In 2021, the reduction was 14.18% and 30.88% on the 7th day and 10th day for S1, respectively, and 11.32% and 25.21% for S3. Physiologically, this reduction coincided with decreased soluble protein content and glutamate-oxaloacetate transaminase and glutamic-pyruvic transaminase activities, alongside increased free amino acid content and protease and peptidase activities. Discussion: We hypothesize that normal night temperatures provide temporary physiological compensation against daytime high-temperature stress damage, maintaining a dynamic balance between Cry1Ac synthesis and degradation. However, as high-temperature stress persists between 4 and 7 days, the cumulative damage exceeds the nighttime compensatory capacity limit, which quantitatively and qualitatively disrupts this metabolic equilibrium by day 7, accelerating endotoxin loss. In summary, prolonged high-temperature stress (approximately 1 week) at the peak square stage in the field environments requires close monitoring and timely agricultural interventions.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.