ArticleJournal of the Endocrine Society2026
Article in Journal of the Endocrine Society, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
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Authors and funding
9 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Context: Unprovoked ketosis-prone type 2 diabetes (KPD) is characterized by a male predominance, young age of onset, and abrupt presentation of diabetic ketosis/ketoacidosis without precipitating factors. Although many individuals regain β-cell function after insulin therapy, some progress to insulin dependence, suggesting overlap with type 1 diabetes. We previously reported the involvement of insulin-associated peptide-specific T helper 1 (Th1) responses in KPD pathogenesis. Exocrine pancreatic inflammation has been documented in type 1 diabetes, with evidence suggesting its contribution to disease development; however, its relevance to KPD remains unclear. Objective: This cross-sectional study aimed to elucidate mechanisms underlying KPD by investigating the association between endocrine and exocrine immune responses. Methods: We recruited 54 participants with KPD, assessed clinical parameters, including human leukocyte antigen ( Results: Conclusion:
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.