Evidence map›Paper›PMID 42698599›Full record

ArticleJournal of traditional and complementary medicine2026

Pharmacological basis for the traditional use of

Najeeb Ur Rehman, Mohd Nazam Ansari, Aman Karim, Abdullah Rabea Tashah, Hussain Mohammad Daak, Khalil Y Abujheisha, Muneeb Ur Rehman, Wasim Ahmad

Abstract read
In one paragraph

Article in Journal of traditional and complementary medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Najeeb Ur RehmanDepartment of Pharmacology and Toxicology, College of Pharmacy, Prince Sattam Bin Abdulaziz University, Al-Kharj, 11942, Kingdom of Saudi Arabia.
Mohd Nazam AnsariDepartment of Pharmacology and Toxicology, College of Pharmacy, Prince Sattam Bin Abdulaziz University, Al-Kharj, 11942, Kingdom of Saudi Arabia.
Aman KarimDepartment of Biotechnology, National University of Medical Sciences (NUMS), Rawalpindi, Pakistan.
Abdullah Rabea TashahDepartment of Pharmacology and Toxicology, College of Pharmacy, Prince Sattam Bin Abdulaziz University, Al-Kharj, 11942, Kingdom of Saudi Arabia.
Hussain Mohammad DaakDepartment of Pharmacology and Toxicology, College of Pharmacy, Prince Sattam Bin Abdulaziz University, Al-Kharj, 11942, Kingdom of Saudi Arabia.
Khalil Y AbujheishaDepartment of Basic Medical Science, Faculty of Medicine and Health Science, Palestine Polytechnic University, Hebron, Palestine.
Muneeb Ur RehmanKey Laboratory for Green Chemical Process of Ministry of Education Hubei Key Laboratory of Novel Reactor and Green Chemical Technology, Hubei Engineering Research Center for Advanced Fine Chemicals, School of Chemical Engineering and Pharmacy Wuhan Institute of Technology, 206 1st Rd Optics Valley, East Lake New Technology Development District, Wuhan, Hubei, 430205, China.
Wasim AhmadDepartment of Pharmacy, Mohammed Al-Mana College for Medical Sciences, Dammam, 34222, Saudi Arabia.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background and aim: This study was carried out to evaluate the possible gut modulatory and antibacterial activities of Experimental procedure: Gas Chromatography-Mass Spectrometry (GC-MS) analysis was conducted for chemical profiling, while FTIR analyzed the chemical bonds. Mice were used for Results and conclusion: GC-MS analysis identified 16 metabolites, while mice administered with lower doses of Ms.Cr (50 and 100 mg/kg) causes increase in the count of wet feces and the total number of fecal outputs, which was significantly reduced in the pre-atropinized mice. At higher doses of 200 and 400 mg/kg, Ms.Cr showed respective protection of 40% and 80% in mice in castor oil induced diarrhea. In jejunal tissues, lower concentrations of Ms.Cr increased the spontaneous contractions followed by complete relaxation at higher concentrations. Further, Ms.Cr caused concentration-dependent (1.0-5.0 mg/mL) relaxation of high K

Indexed as

AntispasmodicCa++ channel blockerMuscarinicMyrica salicifoliaPDE inhibitor

Identifiers

PMID42698599
PMCPMC13541586

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.