ArticleCureus2026
Silent Renal Autodestruction: Incidental Discovery of End-Stage Genitourinary Tuberculosis (Putty Kidney) 14 Years After Treatment for Pulmonary Tuberculosis With Multisystem Sequelae.
Article in Cureus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
3 authors.
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Abstract
Genitourinary tuberculosis (GUTB) is a prevalent yet frequently underdiagnosed form of extrapulmonary tuberculosis (TB), owing to its characteristically indolent and asymptomatic clinical course. In its most advanced form, renal TB culminates in complete destruction and dystrophic calcification of the renal parenchyma, a condition known as "putty kidney" or autonephrectomy. This entity can develop silently over many years, even in patients who have completed antitubercular therapy for pulmonary disease. A 44-year-old woman with a remote history of pulmonary TB treated 14 years prior presented to the gynecology outpatient department for the evaluation of menstrual irregularities. She denied all urinary symptoms. Incidental ultrasonography revealed gross hydronephrosis and calcification of the left kidney. Contrast-enhanced computed tomography (CT) of the abdomen demonstrated a calcified, nonexcreting left kidney consistent with a putty kidney, accompanied by calcified hepatic and splenic granulomas and calcified mesenteric lymph nodes. High-resolution CT of the thorax identified calcified pulmonary nodules consistent with healed pulmonary TB. Magnetic resonance imaging of the spine revealed lytic lesions at L4-L5 suggestive of healed spinal TB. A diethylenetriamine pentaacetic acid (DTPA) renal scan confirmed absent left renal function. The patient underwent open left simple nephrectomy. Histopathological examination demonstrated granulomatous inflammation with multinucleated giant cells and caseous necrosis, confirming renal TB. The postoperative course was uneventful. Renal TB is characterized by slow, progressive hematogenous dissemination of
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