ReviewOncology letters2026
Claudin-18.2: Molecular mechanisms to clinical translation in solid tumors (Review).
Review in Oncology letters, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
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Corrections and comments
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Claudin 18.2 (CLDN18.2) is a functional subtype generated by alternative splicing of the CLDN18 gene. It is aberrantly overexpressed in a variety of gastrointestinal solid tumors such as gastric cancer and pancreatic cancer, and has emerged as a promising target for tumor targeted therapy. To date, zolbetuximab, a monoclonal antibody targeting CLDN18.2, has demonstrated survival benefits in phase III clinical trials. Antibody-drug conjugates, bispecific antibodies and chimeric antigen receptor T cell therapies also exhibit favorable application potential. Nevertheless, multiple challenges remain in this field. Distinction between CLDN18.1 and CLDN18.2 subtypes is often ambiguous, and the lack of unified immunohistochemistry testing criteria, including antibody selection and specimen types, leads to conflicting findings on prognostic research. Additionally, intratumoral heterogeneity, differential functions across tumor types and tumor microenvironment limitations further compromise the efficacy of targeted therapy. As a narrative review, this article systematically elaborates the molecular structure, expression regulatory mechanisms and clinicopathological value of CLDN18.2. Based on the data of 58 global clinical trials, it analyzes the current status, existing problems in testing standardization and drug development. Future directions are proposed, including standardizing detection protocols, developing differentiated drugs, optimizing combination regimens and overcoming drug resistance, aiming to provide references for the clinical translation and standardized application of CLDN18.2-targeted therapies.
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.