Evidence map›Paper›PMID 42619827›Full record

ArticlebioRxiv : the preprint server for biology2026

Genome-wide analysis reveals the importance of histone acetyltransferase Esa1 in transcriptional regulation during nitrogen starvation.

Uzair Khan, Krystal Cvetkovski, Matthew Werick, Cole Tracy, Sara Fatima, Dimitra Dialynaki, Daniel J Klionsky, Chhabi K Govind

Abstract readPreprint
PubMed Full text at PMC Publisher
In one paragraph

Article in bioRxiv : the preprint server for biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Uzair KhanDepartment of Biological Sciences, Oakland University, Rochester, Michigan, USA.
Krystal CvetkovskiDepartment of Biological Sciences, Oakland University, Rochester, Michigan, USA.
Matthew WerickDepartment of Biological Sciences, Oakland University, Rochester, Michigan, USA.
Cole TracyDepartment of Biological Sciences, Oakland University, Rochester, Michigan, USA.
Sara FatimaDepartment of Biological Sciences, Oakland University, Rochester, Michigan, USA.
Dimitra DialynakiLife Sciences Institute and Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI, USA.
Daniel J KlionskyLife Sciences Institute and Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI, USA.ORCID 0000-0002-7828-8118
Chhabi K GovindDepartment of Biological Sciences, Oakland University, Rochester, Michigan, USA.ORCID 0000-0002-6427-6458

Funding

The mechanism and regulation of autophagyR35GM131919 · NIGMS · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI DANIEL J. KLIONSKY · 2019 to 2026
$6.4M
Transcription regulation of recovery from stressR15GM163296 · NIGMS · OAKLAND UNIVERSITY · PI Chhabi K Govind · 2026 to 2026
$566k
Roles for chromatin remodeler RSC and histone acetyltransferases in regulating chromatin structure and transcriptionR15GM148919 · NIGMS · OAKLAND UNIVERSITY · PI GOVIND, CHHABI K · 2022 to 2023
$492k
NIGMS NIH HHS R15 GM148919NIGMS NIH HHS R15 GM163296NIGMS NIH HHS R35 GM131919
6 · The paper itself

Abstract

Macroautophagy/autophagy is a process that degrades intracellular components and is strongly triggered by nitrogen starvation (-N). Some

Indexed as

Amino acid biosynthetic genesATGsAutophagyChIP-seqGcn4histone acetylationhistone acetyltransferasenitrogen starvationribosomal protein genesRNA Pol II

Identifiers

PMID42619827
PMCPMC13484191

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.