ArticleJournal of neurophysiology2026
Large-scale in vivo electrophysiology analysis reveals opposing thalamic and hippocampal excitability after third-trimester-equivalent alcohol exposure in mice.
Mitchell D Morningstar, Gabriela Acosta, Sam A McKenzie, Carlos F Valenzuela, Ben J Clark, David N Linsenbardt
Abstract read
In one paragraphArticle in Journal of neurophysiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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4 · The recordCorrections and comments
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5 · Who and what moneyAuthors and funding
6 authors.
Mitchell D MorningstarDepartment of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, United States.ORCID 0000-0002-3847-192X Gabriela AcostaDepartment of Psychology, University of New Mexico, Albuquerque, New Mexico, United States.
Sam A McKenzieDepartment of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, United States.
Carlos F ValenzuelaDepartment of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, United States.
Ben J ClarkDepartment of Psychology, University of New Mexico, Albuquerque, New Mexico, United States.
David N LinsenbardtDepartment of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, United States.
Funding
Understanding neurophysiological deficits in response inhibition in children with FASDP50AA022534 · NIAAA · UNIVERSITY OF NEW MEXICO HEALTH SCIS CTR · PI Carlos Fernando Valenzuela · 2014 to 2026
$21.5MAlcohol and Developing Neuronal CircuitsR01AA015614 · NIAAA · UNIVERSITY OF NEW MEXICO HEALTH SCIS CTR · PI Carlos Fernando Valenzuela · 2005 to 2026
$3.9MAlcohol and Developing Neuronal CircuitsR37AA015614 · NIAAA · UNIVERSITY OF NEW MEXICO HEALTH SCIS CTR · PI VALENZUELA, CARLOS FERNANDO · 2012 to 2021
$3.5MAlcohol Research Training in NeurosciencesT32AA014127 · NIAAA · UNIVERSITY OF NEW MEXICO HEALTH SCIS CTR · PI Jonathan L Brigman · 2003 to 2026
$3.3MNeurophysiological Genetics of Binge DrinkingR01AA031496 · NIAAA · UNIVERSITY OF NEW MEXICO HEALTH SCIS CTR · PI LINSENBARDT, DAVID NATHANIEL · 2025 to 2025
$2.9MNeural Basis of Spatial Memory Deficits After Prenatal Alcohol ExposureR01AA029700 · NIAAA · UNIVERSITY OF NEW MEXICO · PI Benjamin J Clark · 2022 to 2026
$1.6MLocus coeruleus regulation of hippocampal network reset during learningR01MH140076 · NIMH · UNIVERSITY OF NEW MEXICO HEALTH SCIS CTR · PI Samuel Arnold McKenzie · 2025 to 2026
$1.3MRepeated Binge Drinking and the Genetic Regulation of Corticostriatal SynchronyR00AA025120 · NIAAA · UNIVERSITY OF NEW MEXICO HEALTH SCIS CTR · PI LINSENBARDT, DAVID NATHANIEL · 2020 to 2022
$993kRepeated Binge Drinking and the Genetic Regulation of Corticostriatal SynchronyK99AA025120 · NIAAA · INDIANA UNIVERSITY INDIANAPOLIS · PI LINSENBARDT, DAVID NATHANIEL · 2017 to 2018
$316kHHS | NIH | National Institute of Mental Health (NIMH) MH140076HHS | NIH | National Institute on Alcohol Abuse and Alcoholism (NIAAA) AA014127HHS | NIH | National Institute on Alcohol Abuse and Alcoholism (NIAAA) AA015614HHS | NIH | National Institute on Alcohol Abuse and Alcoholism (NIAAA) AA022534HHS | NIH | National Institute on Alcohol Abuse and Alcoholism (NIAAA) AA025120HHS | NIH | National Institute on Alcohol Abuse and Alcoholism (NIAAA) AA029700HHS | NIH | National Institute on Alcohol Abuse and Alcoholism (NIAAA) AA031496NIAAA NIH HHS K99 AA025120NIAAA NIH HHS P50 AA022534NIAAA NIH HHS R00 AA025120NIAAA NIH HHS R01 AA015614NIAAA NIH HHS R01 AA029700NIAAA NIH HHS R01 AA031496NIAAA NIH HHS R37 AA015614NIAAA NIH HHS T32 AA014127NIMH NIH HHS R01 MH140076
6 · The paper itselfAbstract
Acute, binge-like alcohol exposure during the third-trimester-equivalent period (TTAE) produces apoptotic neurodegeneration in brain regions critical for spatial learning and memory, including the anterior thalamic nuclei (ATN), which are necessary for contextual learning and memory. To investigate the neural mechanisms underlying spatial cognition deficits following developmental alcohol exposure, we performed high-density electrophysiological recordings in and around the ATN of behaving mice, sampling 7,490 neurons across multiple, predominantly thalamic depths. To model acute binge-like TTAE, C57BL/6J mice received two injections of 2.5 g/kg ethanol (or saline) on
Indexed as
Anterior Thalamic NucleiCentral Nervous System DepressantsEthanolFetal Alcohol Spectrum DisordersHippocampusNeuronsPrenatal Exposure Delayed EffectsAction PotentialsAnimalsFemaleMaleMiceMice, Inbred C57BLPregnancyCentral Nervous System DepressantsEthanoldevelopmentFASDhippocampusin vivo electrophysiologythalamus
Identifiers
PMID42616619
PMCPMC13552330
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