Evidence map›Paper›PMID 42614872›Full record

ArticleJournal of cardiovascular echography

Cocaine Induced Cardiomyopathy Presenting with Cardiac Arrest: Insights from Cardiac Magnetic Resonance.

Francesco Mori, Mattia Alberti, Simona Chiusolo, Gabriele Masini, Valentina Barletta, Emanuele Neri, Lorenzo Faggioni, Doralisa Morrone, Raffaele De Caterina, Giovanni Donato Aquaro

Abstract readCase Reports
In one paragraph

Article in Journal of cardiovascular echography. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Francesco Mori *Department of Surgical, Medical and Molecular Pathology and Critical Area, Cardiology Division, University of Pisa, Pisa, Italy.
Mattia Alberti *Department of Surgical, Medical and Molecular Pathology and Critical Area, Cardiology Division, University of Pisa, Pisa, Italy.
Simona ChiusoloDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Cardiology Division, University of Pisa, Pisa, Italy.
Gabriele MasiniDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Cardiology Division, University of Pisa, Pisa, Italy.
Valentina BarlettaDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Second Cardiology Division, University of Pisa, Pisa, Italy.
Emanuele NeriDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Radiology Division, University of Pisa, Pisa, Italy.
Lorenzo FaggioniDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Radiology Division, University of Pisa, Pisa, Italy.
Doralisa MorroneDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Cardiology Division, University of Pisa, Pisa, Italy.
Raffaele De CaterinaDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Cardiology Division, University of Pisa, Pisa, Italy.
Giovanni Donato AquaroDepartment of Surgical, Medical and Molecular Pathology and Critical Area, Radiology Division, University of Pisa, Pisa, Italy.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cocaine abuse is associated with a broad spectrum of cardiovascular complications, including myocardial ischemia, arrhythmias, and cardiomyopathy. However, chronic myocardial injury may remain subclinical, delaying diagnosis until an acute event occurs. We present the case of a 49-year-old man with a history of chronic cocaine use who was admitted following an out-of-hospital cardiac arrest. A post-Return of Spontaneous Circulation Point-of-Care Ultrasound revealed mildly reduced left ventricular systolic function, while coronary angiography excluded obstructive coronary artery disease. Cardiac magnetic resonance (CMR) imaging demonstrated mid-wall late gadolinium enhancement and patchy T1 elevation, consistent with nonischemic myocardial fibrosis and edema. A subcutaneous implantable cardioverter-defibrillator was placed for secondary prevention, and beta-blocker therapy was initiated. Genetic testing for inherited arrhythmogenic and cardiomyopathic conditions was also undertaken. This case underscores the potential for clinically silent cocaine-induced cardiomyopathy and highlights the value of CMR in detecting subclinical myocardial injury.

Indexed as

Arrhythmiascardiac arrestcardiac magnetic resonance imagingcardiomyopathycocaine

Identifiers

PMID42614872
PMCPMC13485265

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.