ArticleEndocrine, metabolic & immune disorders drug targets2026
Association of the
Article in Endocrine, metabolic & immune disorders drug targets, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author.
Funding
No grant is acknowledged in the PubMed record.
Abstract
introductionGenetic polymorphisms affecting oxidative stress and lipid metabolism contribute to type 2 diabetes (T2D). The role of oxidized low-density lipoprotein receptor 1 (
methodsA tertiary hospital-based case-control study was conducted among 143 Saudi adults (≥18 years) in Riyadh from November 2019 to January 2020, including 79 individuals with T2D (fasting blood glucose ≥7 mmol/L) and 64 normoglycemic controls (<5.6 mmol/L). Genomic DNA was extracted from peripheral blood, and the OLR1 rs11053646 SNP was genotyped using PCR-RFLP. Genotype distributions were tested for Hardy-Weinberg equilibrium (HWE). Logistic regression models adjusted for age, sex, and body mass index were used to estimate odds ratios (ORs) with 95% confidence intervals (CIs).
resultsGenotype distribution in controls conformed to HWE (p=0.14). The GG genotype was predominant, while the CC genotype was absent. GC frequency was slightly higher in T2D cases, but not statistically significant. Allele distributions were comparable between groups. Adjusted analysis showed no significant association with T2D risk (OR=0.41, p=0.14). DISCUSSION:
conclusion
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.