ArticleAdvanced science (Weinheim, Baden-Wurttemberg, Germany)2026
Ferroptosis Suppression by the M
Article in Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Authors and funding
11 authors.
Funding
Abstract
aimsPancreatic ductal adenocarcinoma (PDAC) is a lethal malignancy with poor prognosis, and gemcitabine-based chemotherapy remains the standard first-line treatment. However, the rapid emergence of chemoresistance limits its efficacy. Elucidating the molecular mechanisms underlying gemcitabine resistance is critical for improving therapeutic outcomes. APPROACHES &
resultsBy integrating transcriptomic profiling of gemcitabine-resistant PDAC cells, patient-derived xenografts (PDXs), and pretreatment clinical biopsy specimens, we identified the m
conclusionsCollectively, this study uncovers a previously unrecognized epitranscriptomic mechanism linking m
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.