ReviewNeurobiology of pain (Cambridge, Mass.)
Pain in the claustrum.
Review in Neurobiology of pain (Cambridge, Mass.). The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
6 authors.
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Abstract
Pain arises from coordinated activity across distributed brain networks. The claustrum, a thin subcortical nucleus with extensive bidirectional connectivity with the neocortex, has recently emerged as a potential hub in this network. Although historically difficult to isolate due to its thin anatomical structure and proximity to other pain-associated regions, recent advances in neuroimaging and circuit-specific manipulations have revealed new insights into its role in pain. This review synthesizes the evidence from human and animal studies and evaluates the evidential strength of current approaches, ranging from incidental reporting in whole-brain neuroimaging studies to claustrum-targeted imaging, electrophysiology and projection-specific manipulations. The literature suggests that the claustrum contributes to pain not as a primary sensory relay, but as a modulatory and integrative structure influencing various facets of cognition. We have organized these findings into three theory-guided frameworks: (1) sensory filtering of salient stimuli, (2) cognitive network engagement, and (3) offline consolidation of pain-related memory during rest and sleep. In chronic pain, claustrocortical function is altered in ways that map onto each of these frameworks, with rodent models pointing to depressed claustrocingulate output and loss of inhibitory control, and human imaging linking claustrum signals to catastrophizing, expectancy, and altered network recruitment. Taken together, the evidence highlights the claustrum as a promising but incompletely understood node in pain circuitry and underscores the need for more anatomically precise and mechanistically decisive studies to define its contribution to normal and pathological pain states.
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.