ArticleAdvanced science (Weinheim, Baden-Wurttemberg, Germany)2026
Rhoifolin Alleviates Ulcerative Colitis by Targeting NMNAT1 and Activating SIRT1-FOXO1 Signaling to Enhance ILC3s Effector Function.
Article in Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
13 authors.
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Abstract
Ulcerative colitis (UC) is a chronic inflammatory bowel disorder with limited therapeutic options, particularly those capable of selectively reinforcing intestinal epithelial barrier integrity while preserving immune homeostasis. Here, Rhoifolin (3, 10 mg/kg) dose-dependently alleviates DSS and TNBS-induced colitis in mice. Mechanistically, Rhoifolin selectively potentiates ILC3s effector function without affecting their proliferation, apoptosis, intestinal homing, developmental differentiation, or the distribution of CCR6
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