ArticleInflammation research : official journal of the European Histamine Research Society ... [et al.]2026
NKR
Article in Inflammation research : official journal of the European Histamine Research Society ... [et al.], 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
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0 citing papers in PubMed.
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Authors and funding
6 authors.
Funding
Abstract
backgroundInterferon-gamma (IFNγ) is a pivotal cytokine in immune surveillance and inflammation, yet its precise role in colorectal cancer (CRC) remains controversial due to its dual involvement in tumor suppression and immune evasion. Although natural killer cell receptor (NKR)
methodsAn azoxymethane/dextran sodium sulfate (AOM/DSS)-induced CRC model was established in heterozygous Yeti mice with IFNγ-mediated hyperinflammation. Using flow cytometry and histology, we evaluated tumor burden, histopathology, and immune cell infiltration in tumor tissues. NKR
resultsWe found that Yeti mice display significantly blunted CRC development compared to wild-type (WT) controls. Furthermore, these IFNγ-mediated anti-tumor immune responses were attributed to increased tumor infiltration by CD8
conclusionsNKR
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42536164What OpenQuestion holds
Registered trials
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