Evidence map›Paper›PMID 42529647›Full record

ArticleFrontiers in cellular and infection microbiology2026

Whole genome sequencing of ceftolozane/tazobactam-resistant, XDR

Samira M Hamed, Amira F A Hussein, Moshira H Ezz El Arab, Mohamed H Al-Agamy, Mohammed Aufy, Mohamed Abdelmoteleb, Mai M Zafer

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Article in Frontiers in cellular and infection microbiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Samira M HamedDepartment of Microbiology and Immunology, Faculty of Pharmacy, October University for Modern Sciences and Arts (MSA), Giza, Egypt.
Amira F A HusseinClinical and Chemical Pathology Department, Faculty of Medicine, Cairo University, Cairo, Egypt.
Moshira H Ezz El ArabClinical and Chemical Pathology Department, Faculty of Medicine, Cairo University, Cairo, Egypt.
Mohamed H Al-AgamyDepartment of Pharmaceutics, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
Mohammed AufyDepartment of Pharmaceutical Sciences, Division of Pharmacology and Toxicology, University of Vienna, Vienna, Austria.
Mohamed AbdelmotelebBotany Department, Faculty of Science, Mansoura University, Mansoura, Egypt.
Mai M ZaferDepartment of Microbiology and Immunology, Faculty of Pharmacy, Ahram Canadian University, Cairo, Egypt.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Introduction: Ventilator-associated pneumonia (VAP) caused by Methods: In this study, we assessed the antimicrobial susceptibility profiles of 42 Results: All five isolates belonged to serogroup O11 and sequence type ST773. Nevertheless, WGS-based phylogenetic analyses, such as core genome MLST and SNP-based phylogeny, showed that the isolates were non-clonal and had a closer genetic relationship to previously identified ST773 strains from Egypt and Germany. Several multidrug efflux systems and a broad range of acquired antimicrobial resistance genes, such as Discussion: This study reports the identification of non-clonal, XDR

Indexed as

Anti-Bacterial AgentsCephalosporinsDrug Resistance, Multiple, BacterialPneumonia, Ventilator-AssociatedPseudomonas aeruginosaPseudomonas InfectionsTazobactamChild, PreschoolCritical IllnessDrug CombinationsGenome, BacterialHumansInfantInfant, NewbornIntensive Care Units, PediatricMicrobial Sensitivity TestsAnti-Bacterial Agentsceftolozane, tazobactam drug combinationCephalosporinsDrug CombinationsTazobactamThird Generation CephalosporinsExtensive drug resistanceNDMPseudomonas aeruginosaresistance islandsST773ventilator-associated pneumoniawhole genome sequencing

Identifiers

PMID42529647
PMCPMC13417825

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.