ArticleEuropean journal of applied physiology2026
Running in the heat reduces time to task failure, attenuates biomarkers of muscle microtrauma, and exacerbates low-frequency force depression and cardiac vagal withdrawal over 72-h post-exercise.
Article in European journal of applied physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
purposeAs exercise in the heat becomes increasingly common, a better understanding of post-exercise responses is essential to guide the appropriate use of fatigability monitoring tools and recovery strategies. Therefore, this study investigated the effect of running in the heat on neuromuscular function, cardiac autonomic modulation, and plasma markers of muscle microtrauma during post-exercise recovery.
methodsTwelve participants (V̇O
resultsRunning in the heat reduced time to task failure (TTF, 17.8 ± 4.4 min vs. 42.3 ± 20.1, p < 0.01, d = 1.96) and attenuated the increase in plasma CK at 24 h post-exercise (condition×time interaction, post-hoc p < 0.05). Neuromuscular function and cardiac autonomic modulation were impaired within the first hour post-exercise (time effects, p < 0.05) and returned to baseline values at 24-h post-exercise in both conditions (post-hoc p > 0.05). Aggregated data (condition effects, post-hoc p < 0.05) over the 72-h monitoring time evidenced exacerbation of low-frequency force depression (reduced Db10
conclusionRunning in the heat at vRCP reduced TTF. Although heat reduced exercise duration, it specifically exacerbated low-frequency force depression and cardiac vagal withdrawal for 72-h post-exercise while attenuating the increase in plasma markers of muscle microtrauma at 24-h post-exercise.
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