ArticleObesity surgery2026
Longitudinal Evolution of Gastric Sleeve Anatomy and its Association with Esophagitis Progression Following Laparoscopic Sleeve Gastrectomy.
Article in Obesity surgery, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
15 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
introductionLaparoscopic Sleeve Gastrectomy (LSG) is the most common bariatric procedure, effective for weight loss and comorbidity resolution. However, post-LSG Gastroesophageal Reflux Disease (GERD) and endoscopic esophagitis (EE) are significant long-term concerns, often linked to anatomical sleeve changes. Longitudinal data assessing these alterations and their association with EE progression remain limited.
methodsThis retrospective study included 539 of 754 LSG patients who underwent at least one post-operative endoscopy at a tertiary bariatric center. Demographic, clinical, and endoscopic data were analyzed. Longitudinal comparisons were performed among patients who had endoscopies at both 1-4 and ≥ 5 years post-LSG using McNemar's test, with logistic regression to identify predictors of EE.
resultsAt 1-4 years post-LSG (N = 505), EE was detected in 22.0% and GERD symptoms in 30.1%. Anatomical findings included sleeve dilatation (5.9%), hiatal hernia (HH) (7.9%), twisting (3.7%), and narrowing (0.8%), with composite abnormalities in 16.2%. GERD symptoms independently predicted early EE (OR 1.86, 95% CI 1.19-2.90, p = 0.003). At ≥ 5 years (N = 117), EE increased to 28.2% and GERD symptoms to 65.0%. In the paired subgroup (N = 83), the prevalence of GERD symptoms rose from 10.8% to 67.5% (p < 0.001), composite anatomical findings from 16.9% to 39.5% (p = 0.009), and sleeve dilatation from 7.2% to 25.3% (p = 0.004). However, early GERD, EE, HH, and composite findings did not independently predict late EE.
conclusionLong-term GERD and EE are common and progressive after LSG, with increasing anatomical changes over time. Early post-operative findings fail to predict late disease, highlighting the multifactorial evolution of post-LSG reflux and the need for ongoing surveillance.
Indexed as
Identifiers
42477225What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.