ArticleBiochemistry and biophysics reports2026
RL-QN15 accelerates vaginal mucosal repair by regulating inflammatory, autophagic, and apoptotic responses.
Article in Biochemistry and biophysics reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Background: Vaginal mucosal injury, caused by infection, trauma, or estrogen deficiency, poses a significant clinical challenge with limited effective therapies. Autophagy and apoptosis are critical regulators of mucosal repair, yet their modulation by therapeutic peptides remains poorly understood. Objective: This study aimed to investigate whether the amphibian-derived peptide RL-QN15 promotes vaginal mucosal repair and to elucidate its underlying mechanisms. Methods: The therapeutic efficacy and mechanisms of RL-QN15 were investigated in both Results: RL-QN15 significantly reversed LPS-induced suppression of proliferation and autophagy, while inhibiting apoptosis and IL-6 expression Conclusions: RL-QN15 promotes vaginal mucosal regeneration by modulating autophagy, apoptosis and alleviating inflammation, positioning it as a promising multitarget peptide therapeutic for mucosal injury.
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