ArticleScientific reports2026
Neuronal expression of retinoid-related orphan receptor gamma (RORγ) and revisiting its role in the central nervous system.
Article in Scientific reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Retinoid-related orphan receptor gamma (RORγ) is a lineage-defining transcription factor for T helper 17 (Th17) cells and has long been considered selectively expressed within the immune system. However, accumulating evidence has implicated Th17 cells and other RORγ-expressing immune populations in embryonic brain development and central nervous system (CNS) function. Notably, RORγ-Cre-mediated deletion of the tuberous sclerosis complex (TSC) has been reported to cause spontaneous seizures and premature lethality, leading to the conclusion that RORγ-expressing immune cells directly regulate CNS physiology. Using multiple RORγ-based reporter mouse lines, we identified unexpected and widespread neuronal labeling throughout the forebrain and cerebellum. RORγ-Cre: GFP
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