In one paragraphArticle in International journal of molecular sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
10 authors.
Vanessa Ruiz-Esparza-PalaciosDepartment of Bioengineering, School of Engineering, Tecnologico de Monterrey, Campus Ciudad de Mexico, Mexico City 14380, Mexico.ORCID 0000-0003-2067-9316 Ian García-AguirreDepartment of Bioengineering, School of Engineering, Tecnologico de Monterrey, Campus Ciudad de Mexico, Mexico City 14380, Mexico.ORCID 0000-0002-3903-1467 Guadalupe E Jiménez-GutiérrezLaboratory of Genomic Medicine, Department of Genetics, National Rehabilitation Institute-Luis Guillermo Ibarra Ibarra (INR-LGII), Mexico City 14389, Mexico.
Nadia M Murillo-MeloDepartment of Bioengineering, School of Engineering, Tecnologico de Monterrey, Campus Ciudad de Mexico, Mexico City 14380, Mexico.
Aranza Meza-DorantesDepartment of Bioengineering, School of Engineering, Tecnologico de Monterrey, Campus Ciudad de Mexico, Mexico City 14380, Mexico.ORCID 0000-0003-4483-371X Yessica S Tapia-GuerreroLaboratory of Genomic Medicine, Department of Genetics, National Rehabilitation Institute-Luis Guillermo Ibarra Ibarra (INR-LGII), Mexico City 14389, Mexico.ORCID 0000-0003-3281-4757 Oscar Pérez-MéndezDepartment of Bioengineering, School of Engineering, Tecnologico de Monterrey, Campus Ciudad de Mexico, Mexico City 14380, Mexico.ORCID 0000-0002-6977-1829 Jose M Gonzalez-MeljemDepartment of Bioengineering, School of Engineering, Tecnologico de Monterrey, Campus Ciudad de Mexico, Mexico City 14380, Mexico.ORCID 0000-0002-1554-0363 Bulmaro CisnerosDepartment of Genetics and Molecular Biology, Center of Research and Advanced Studies (CINVESTAV-IPN), Mexico City 07360, Mexico.ORCID 0000-0003-2802-1201 Jonathan J MagañaDepartment of Bioengineering, School of Engineering, Tecnologico de Monterrey, Campus Ciudad de Mexico, Mexico City 14380, Mexico.ORCID 0000-0003-4399-4618 Funding
Challenge-Based Research Funding Program ITESM CI_EIC_HLT_D_208SECIHTI 1078454SECIHTI CBF-2025-G-1656
6 · The paper itselfAbstract
Spinocerebellar ataxia type 7 (SCA7) is a hereditary disorder characterized by degeneration of the cerebellum and retina. SCA7 is caused by the expansion of a polyQ tract in the
Indexed as
Cellular SenescenceDNA DamageEpendymoglial CellsNuclear LaminaSpinocerebellar AtaxiasAnimalsAtaxin-7Genomic InstabilityHumansPeptidesAtaxin-7ATXN7 protein, humanPeptidespolyglutaminecellular senescenceDNA damageMüller glial cellspolyglutamine diseasesspinocerebellar ataxia type 7
Identifiers
PMID42449987
PMCPMC13361445
What OpenQuestion holds
Textmetadata
LicenceCC BY
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