ReviewArquivos de neuro-psiquiatria2026
Epilepsy and amygdalar enlargement: cause or consequence?
Review in Arquivos de neuro-psiquiatria, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
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Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Abstract: The amygdala is a central structure of the limbic system. For several decades, volumetric asymmetries of the amygdala have been described, initially associated with psychiatric disorders. In epilepsy, a growing literature highlights the distinctive features of amygdala enlargement (AE), considering that since its original description, diagnosis has been primarily radiological. The objective of the present work is to demonstrate, through reviews of existing academic publications and through our experience, that its function goes beyond imaging, contemplating a defined clinical, electrographic, and histopathological pattern that responds to both medical and surgical treatment. Therefore, it would be an entity that extends beyond a reactive phenomenon. In conclusion, AE would be a specific syndrome defined with particular characteristics that extends beyond a reactive mechanism secondary to adjacent epileptogenic networks.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.