Evidence map›Paper›PMID 42439625›Full record

ArticleCells2026

Extra-Neurological Characterization of Seckel Syndrome-Model Mice Harboring

Nanako Hamada, Koki Ichihashi, Tohru Matsuki, Ikuko Iwamoto, Atsuo Nakayama, Akira Hara, Koh-Ichi Nagata

Abstract read
In one paragraph

Article in Cells, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Nanako HamadaDepartment of Molecular Neurobiology, Institute for Developmental Research, Aichi Developmental Disability Center, Kasugai 480-0392, Japan.
Koki IchihashiDepartment of Tumor Pathology, Gifu University School of Medicine, Gifu 501-1194, Japan.ORCID 0009-0002-5373-7471
Tohru MatsukiDepartment of Cellular Pathology, Institute for Developmental Research, Aichi Developmental Disability Center, Kasugai 480-0392, Japan.ORCID 0000-0003-1832-2406
Ikuko IwamotoDepartment of Molecular Neurobiology, Institute for Developmental Research, Aichi Developmental Disability Center, Kasugai 480-0392, Japan.
Atsuo NakayamaDepartment of Cellular Pathology, Institute for Developmental Research, Aichi Developmental Disability Center, Kasugai 480-0392, Japan.ORCID 0000-0003-1444-0800
Akira HaraDepartment of Tumor Pathology, Gifu University School of Medicine, Gifu 501-1194, Japan.
Koh-Ichi NagataDepartment of Molecular Neurobiology, Institute for Developmental Research, Aichi Developmental Disability Center, Kasugai 480-0392, Japan.ORCID 0000-0002-6827-8434

Funding

Daiko Foundation 9234Japan Foundation for Pediatric Research N/AJapan Society for the Promotion of Science 23K07284Japan Society for the Promotion of Science JP20K21589Japan Society for the Promotion of Science JP22H03049
6 · The paper itself

Abstract

Centrosomal protein 152 (CEP152) is a key regulator of centriole architecture and function, essential for proper cell division and polarity. Pathogenic variants in

Indexed as

Anemia, MacrocyticCell Cycle ProteinsCentrosomal Associated ProteinsDwarfismMicrocephalyAnimalsDisease Models, AnimalFaciesHumansMaleMiceMutationPhenotypeSpermatogenesisCell Cycle ProteinsCentrosomal Associated ProteinsCEP152macrocytic anemiamouse modelspathogenic variantsSeckel syndrometestis development

Identifiers

PMID42439625
PMCPMC13359663

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.