Evidence map›Paper›PMID 42421798›Full record

ArticleJournal of toxicology2026

Fenpropathrin, A Pyrethroid Pesticide, Induces Dopaminergic Neurodegeneration in

Saba Afsheen, Ahmed Shaney Rehman, Swati Chandra, Mohammad Mumtaz Alam, Suhel Parvez

Abstract read
In one paragraph

Article in Journal of toxicology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Saba AfsheenDepartment of Toxicology, School of Chemical and Life Sciences, Jamia Hamdard, New Delhi, 110062, India, jamiahamdard.edu.ORCID https://orcid.org/0009-0008-6707-0667
Ahmed Shaney RehmanDepartment of Toxicology, School of Chemical and Life Sciences, Jamia Hamdard, New Delhi, 110062, India, jamiahamdard.edu.ORCID https://orcid.org/0009-0005-8636-2241
Swati ChandraDepartment of Toxicology, School of Chemical and Life Sciences, Jamia Hamdard, New Delhi, 110062, India, jamiahamdard.edu.
Mohammad Mumtaz AlamDepartment of Pharmaceutical Chemistry, School of Pharmaceutical Education and Research, Jamia Hamdard, New Delhi, 110062, India, jamiahamdard.edu.ORCID https://orcid.org/0000-0001-6420-8757
Suhel ParvezDepartment of Toxicology, School of Chemical and Life Sciences, Jamia Hamdard, New Delhi, 110062, India, jamiahamdard.edu.ORCID https://orcid.org/0000-0002-6318-6506

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Parkinson's disease (PD) is a neurodegenerative condition that typically develops as people age and is characterized by the progressive death of dopaminergic neurons in the substantia nigra area of the midbrain. The etiology of PD includes both genetic and environmental components. Epidemiological studies have shown that pesticide exposure is an important contributory risk factor for PD. Fenpropathrin has been shown to have neurotoxic effects on rodents, but the exact mechanism and its ability to replicate the pathological features associated with PD remain unclear. Therefore, the present study investigated the neurotoxic effects of Fenpropathrin in

Indexed as

Drosophila melanogasterfenpropathrinneurodegenerationParkinson’s diseasetyrosine hydroxylase

Identifiers

PMID42421798
PMCPMC13343313

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.