Article in JCPP advances, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
The Early Growth and Development Study Pediatric CohortUH3OD023389 · OD · UNIVERSITY OF OREGON · PI Jody M. Ganiban, Leslie Diane Leve · 2018 to 2026
$16.6M
The Early Growth and Development Study: Family Process, Genes and School EntryR01HD042608 · NICHD · OREGON SOCIAL LEARNING CENTER, INC. · PI CAPALDI, DEBORAH M. · 2002 to 2011
$14.9M
The Early Growth and Development Study Pediatric CohortUG3OD023389 · OD · UNIVERSITY OF OREGON · PI GANIBAN, JODY M., LEVE, LESLIE DIANE · 2016 to 2024
$7.5M
Genes, Prenatal Drug Exposure, and the Postnatal Environment: An Adoption StudyR01DA020585 · NIDA · PENNSYLVANIA STATE UNIVERSITY, THE · PI NEIDERHISER, JENAE M. · 2007 to 2011
$5.9M
An adoption study of the development of early substance use: the joint roles of genetic influences, prenatal risk, rearing environment, and pubertal maturationR01DA045108 · NIDA · PENNSYLVANIA STATE UNIVERSITY, THE · PI NEIDERHISER, JENAE M. · 2018 to 2022
$3.7M
Mechanisms underlying resilience to neighborhood disadvantage (Administrative Supplement)UH3MH114249 · NIMH · MICHIGAN STATE UNIVERSITY · PI BURT, S. ALEXANDRA, HYDE, LUKE WILLIAMSON · 2018 to 2022
$3.5M
Neurobiological pathways underlying maladaptive behaviors in youthR01HD093334 · NICHD · MICHIGAN STATE UNIVERSITY · PI BURT, S. ALEXANDRA, HYDE, LUKE WILLIAMSON · 2017 to 2021
$3.2M
Gene-Environment Interplay and Development of Psychiatric Symptoms in ChildrenR01MH092118 · NIMH · PENNSYLVANIA STATE UNIVERSITY, THE · PI LEVE, LESLIE DIANE, NEIDERHISER, JENAE M. · 2010 to 2014
$3.1M
Integrating contextual, proximal, and individual risks for child conduct problemsR01HD066040 · NICHD · MICHIGAN STATE UNIVERSITY · PI BURT, S. ALEXANDRA · 2010 to 2014
$2.9M
Gene-Environment interplay and childhood obesity: An Adoption study.R01DK090264 · NIDDK · GEORGE WASHINGTON UNIVERSITY · PI GANIBAN, JODY M. · 2011 to 2015
$2.8M
Siblings Reared Apart: A Naturalistic Cross-Fostering Study of Young ChildrenR01DA035062 · NIDA · UNIVERSITY OF OREGON · PI LEVE, LESLIE DIANE · 2013 to 2016
$2.7M
Gene-environment Interactions in Childhood Conduct ProblemsR01MH081813 · NIMH · MICHIGAN STATE UNIVERSITY · PI BURT, S. ALEXANDRA · 2008 to 2013
Background: Multiple dimensions of parenting-including hostility, conflict, and warmth-are associated with an array of mental disorders in youth. We tested two possible explanations for these associations: Parenting may shape a shared liability to experience many forms of psychopathology ("shared-liability" hypothesis), or parenting may shape unique liabilities to externalizing and internalizing psychopathology ("unique-liability" hypothesis). Methods: Participants were from the Early Growth and Development Study (EGDS; Results: Across ages and cohorts, parenting characterized by hostility and conflict was associated with greater p, consistent with the shared-liability hypothesis. Also consistent, parental warmth was associated with lower p in middle and late childhood in EGDS participants, though parental involvement was not associated with p in adolescent MTwiNS participants. There was limited support for the unique-liability hypothesis; only one association (with externalizing psychopathology) remained after accounting for p. In childhood, but not adolescence, findings remained after accounting for genetic confounding. Conclusion: Parenting behaviors, particularly hostility and conflict, are associated with children's broad vulnerability to psychopathology, and these associations are evident in childhood even after controlling for genetic influences. These findings support parenting interventions as a means to reduce children's liability to diverse mental-health problems.
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Disentangling shared and unique effects of parenting on psychopathology: Evidence from two prospective, genetically sensitive cohort studies. · full record | OpenQuestion