ReviewJACC. Asia2026
Applied Anatomy of Perimembranous Ventricular Septal Defect for Transcatheter Device Closure.
Review in JACC. Asia, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Perimembranous ventricular septal defect is a common congenital heart disease effectively treated by transcatheter device closure with high success rates and minimal complications. This review categorizes perimembranous ventricular septal defects into 7 morphologic variants based on anatomical extension and correlates these with echocardiographic classifications: type 1 is central perimembranous; type 2 is superior-anterior extension; type 3 is superior-posterior extension; type 4 is inferior-anterior extension; type 5 is inferior-posterior extension; type 6 is confluent extensions with more than one extension direction; and type 7 is complex type with outlet septum involvement. Type 2 defects are often associated with aortic valve prolapse and regurgitation, requiring soft, flexible devices to minimize valve injury. Type 5 defects lie close to the conduction system; deploying devices within an aneurysmal pouch helps prevent heart block and tricuspid regurgitation. Type 6 and 7 defects sometimes necessitate multiple devices for complete closure. Understanding these anatomical variants aids in selecting appropriate devices and optimizing procedural outcomes.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.