ArticleWorld journal of nephrology2026
Protective effect of boric acid on kidney tissue and oxidative stress after testicular torsion/detorsion in male rats.
Article in World journal of nephrology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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5 authors.
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Abstract
backgroundTesticular ischemia/reperfusion (I/R) injury may affect distant organs. During I/R, metabolites formed in the affected organ are released into the circulation; thus, non-ischemic organs, such as the kidneys and lungs, become involved in the inflammatory process, even if they are not primarily affected. Damage from testicular torsion/detorsion (T/D) models has been shown to affect kidneys. There is a need to examine the nephroprotective effect of boric acid (BA), which is known to have antioxidant effects, on kidney damage.
aimTo investigate kidney damage in a rat testicular T/D model and the effectiveness of BA on this distant organ damage.
methodsTwenty-four male Wistar albino rats were divided into four groups (
resultsAn increase in MDA levels and a decrease in PON-1 activity were detected in the I/R group compared to the control group (
conclusionThese findings show that the testicular T/D model in rats causes kidney damage, and BA administration reduces this damage in the kidneys.
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