ArticleOpen veterinary journal2026
Molecular characterization of
Article in Open veterinary journal, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Background: Neuroinflammation can be triggered by traumatic brain injury (TBI) and exposure to toxicants such as chlorpyrifos (CPF), leading to chronic disabilities and substantial health care costs. CD59 and glial maturation factor-beta proteins are implicated in various brain regulatory mechanisms. Investigating their roles can provide valuable insights into their role in pathologies such as TBI. Aim: This study aimed to analyze the molecular characteristics of the Methods: Thirty Results: Both genes exhibited conserved features compared with other fish species. In the brain injury model, fish subjected to brain injury showed reduced expression of Conclusion: Downregulation of CD59 after brain injury suggests a loss of immunomodulatory capacity following brain damage. In CPF-exposed fish, the overexpression of both
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