Evidence map›Paper›PMID 42283474›Full record

ArticleMicrobiology spectrum2026

Identification of genes and mutations involved in antimicrobial low susceptibility in

Eiji Hata

Abstract read
In one paragraph

Article in Microbiology spectrum, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Eiji HataArthropod-borne Disease Group, Department of Transboundary Animal Diseases, National Institute of Animal Health (NIAH), National Agriculture and Food Research Organization (NARO), Kagoshima, Kagoshima, Japan.ORCID 0000-0002-8278-7315

Funding

Ministry of Agriculture, Forestry and Fisheries JPJ008617.17935699
6 · The paper itself

Abstract

IMPORTANCE: Antimicrobial susceptibility testing for mycoplasma using conventional culture methods takes a long time due to the slow culture rate. This has been a major problem in selecting effective antibacterial agents for antimicrobial therapy. Therefore, we identified the genes and mutations involved in decreased susceptibility and developed a genetic susceptibility determination method that can directly detect them. This method makes it possible to quickly and easily detect the antimicrobial susceptibility characteristics of mycoplasmas, which have been difficult to understand until now. In recent years, the emergence and spread of low-susceptibility field strains of various bovine mycoplasmas has been confirmed, raising concerns about this situation. Utilizing this method is expected to improve cure rates and reduce the use of antimicrobial agents.

Indexed as

Anti-Bacterial AgentsDrug Resistance, BacterialMycoplasmaMycoplasma InfectionsAnimalsBacterial ProteinsCattleCattle DiseasesDNA GyraseDNA Topoisomerase IVJapanMicrobial Sensitivity TestsMutationRNA, Ribosomal, 23SAnti-Bacterial AgentsBacterial ProteinsDNA GyraseDNA Topoisomerase IVRNA, Ribosomal, 23Santibiotic resistancegenetic determination of antimicrobial susceptibilityMycoplasma bovirhinisprophage

Identifiers

PMID42283474
PMCPMC13340128

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.