ArticleRedox biochemistry and chemistry2025
Peroxidation rate constants and mechanisms of isoprenoid-derived lipids and their roles in ferroptosis.
Article in Redox biochemistry and chemistry, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
1 citing paper in PubMed.
- Nano-formulations in therapeutic strategies targeting cancer ferroptosis.Journal of pharmaceutical analysis · 2026Review
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Authors and funding
2 authors.
Funding
Abstract
Ferroptosis is a type of regulated cell death that is dependent on iron and driven by lipid peroxidation. Polyunsaturated fatty acids (PUFAs) sensitize cells to ferroptosis as they are prone to lipid peroxidation while monounsaturated fatty acids confer resistance to ferroptosis when incorporated into the lipid membrane as they are much less reactive toward lipid peroxidation. Recently, in addition to fatty acid-derived lipids, isoprenoid-derived lipids have been found to regulate ferroptosis. Specifically, ferroptosis suppressor protein 1 (FSP1) was found to be anti-ferroptotic as it reduces the oxidized forms of coenzyme Q
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Registered trials
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