Evidence map›Paper›PMID 42254865›Full record

ArticleFrontiers in molecular neuroscience2026

Mass spectrometry-based proteomic profiling of human tauopathy brains suggests mitochondria-associated alterations.

Alfi Raudatil Jannah, Mai Hasegawa, Jonathan Ham, Norikazu Hara, Tamao Tsukie, Ai Obinata, Masataka Kikuchi, Kensaku Kasuga, Haruyasu Yamaguchi, Hideomi Hamasaki and 5 more

Abstract read
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In one paragraph

Article in Frontiers in molecular neuroscience, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

15 authors.

Alfi Raudatil Jannah *Department of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Mai Hasegawa *Department of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Jonathan Ham *Department of Pathology, Brain Research Institute, Niigata University, Niigata, Japan.
Norikazu Hara *Department of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Tamao TsukieDepartment of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Ai ObinataDepartment of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Masataka KikuchiDepartment of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Kensaku KasugaDepartment of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Haruyasu YamaguchiGunma University, Maebashi, Japan.
Hideomi HamasakiDepartment of Pathology, Brain Research Institute, Niigata University, Niigata, Japan.
Mari TadaDepartment of Pathology, Brain Research Institute, Niigata University, Niigata, Japan.
Akiyoshi KakitaDepartment of Pathology, Brain Research Institute, Niigata University, Niigata, Japan.
Masaki MatsumotoDepartment of Omics and Systems Biology, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan.
Akinori MiyashitaDepartment of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.
Takeshi IkeuchiDepartment of Molecular Genetics, Brain Research Institute, Niigata University, Niigata, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Tauopathies are neurodegenerative disorders characterized by intracellular accumulation of abnormal tau encoded by

Indexed as

Alzheimer’s diseasecorticobasal degenerationmass spectrometryprogressive supranuclear palsysnRNA-seqtautauopathiestauopathy

Identifiers

PMID42254865
PMCPMC13236917

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.