ArticleParticle and fibre toxicology2026
Targeting the PPAR-γ-ABC transporter axis ameliorates secondary pulmonary alveolar proteinosis induced by indium oxide nanoparticles.
Article in Particle and fibre toxicology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
backgroundPulmonary alveolar proteinosis (PAP) is a rare lung disease with primary (usually autoimmune) and secondary forms. Unlike the autoimmune type, which can be treated with cytokine therapy or whole-lung lavage, the secondary form associated with occupational particulate exposure, such as indium compounds, has no established therapy and remains challenging to treat.
resultsPAP was experimentally induced in rats through intratracheal exposure to indium oxide nanoparticles (In
conclusionTargeting PPAR-γ-dependent macrophage lipid regulation may serve to attenuate In
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