ReviewCommunications medicine2026
Emerging signal transduction pathways and the role of bacterial extracellular vesicles linked to the pathogenesis of endometriosis.
Review in Communications medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
3 authors.
Funding
Abstract
Endometriosis is defined as the ectopic proliferation of endometrial cells. Aberrant signal transduction is present in ectopic endometriotic lesions, and bacteria also contributes to the development of endometriosis by transmitting pathogenic signals through bacterial extracellular vesicles (BEVs). Here, we review the relationship between endometriosis and signal transduction, including classical hormonal, inflammatory, and angiogenic signaling pathways, as well as emerging microbial and epigenetic pathways. We further discuss future challenges from the perspective of molecular signal transduction tools such as extracellular vesicles. Improved understanding of signal transduction in endometriosis will be valuable to develop novel therapies for endometriosis.
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Registered trials
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