Evidence map›Paper›PMID 42225719›Full record

ArticleScientific reports2026

Resistance of E2F4DN to p38

Aina M Llabrés-Mas, Noelia López-Sánchez, Alberto Garrido-García, Vanesa Cano-Daganzo, Irene Camacho-Olmos, José María Frade

Abstract read
In one paragraph

Article in Scientific reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Aina M Llabrés-MasDepartment of Molecular, Cellular and Developmental Neurobiology, Cajal Institute, Consejo Superior de Investigaciones Científicas, 28002, Madrid, Spain.
Noelia López-SánchezDepartment of Molecular, Cellular and Developmental Neurobiology, Cajal Institute, Consejo Superior de Investigaciones Científicas, 28002, Madrid, Spain.
Alberto Garrido-GarcíaDepartment of Molecular, Cellular and Developmental Neurobiology, Cajal Institute, Consejo Superior de Investigaciones Científicas, 28002, Madrid, Spain.
Vanesa Cano-DaganzoDepartment of Molecular, Cellular and Developmental Neurobiology, Cajal Institute, Consejo Superior de Investigaciones Científicas, 28002, Madrid, Spain.
Irene Camacho-OlmosDepartment of Molecular, Cellular and Developmental Neurobiology, Cajal Institute, Consejo Superior de Investigaciones Científicas, 28002, Madrid, Spain.
José María FradeDepartment of Molecular, Cellular and Developmental Neurobiology, Cajal Institute, Consejo Superior de Investigaciones Científicas, 28002, Madrid, Spain. frade@cajal.csic.es.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

E2F4 is a transcription factor involved in cellular homeostasis and a substrate of the stress-activated kinase p38

Indexed as

DNA DamageE2F4 Transcription FactorNeuronsp38 Mitogen-Activated Protein KinasesAnimalsApoptosisCamptothecinCaspase 3Cell Line, TumorE2F1 Transcription FactorMicePhosphorylationCamptothecinCaspase 3E2F1 Transcription FactorE2F4 Transcription Factorp38 Mitogen-Activated Protein KinasesCamptothecinCell stressE2F4HomeostasisPhosphorylation

Identifiers

PMID42225719
PMCPMC13457616

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.