In one paragraphArticle in American journal of physiology. Heart and circulatory physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
5 · Who and what moneyAuthors and funding
8 authors.
Colin W MeansDepartment of Biomedical Engineering, Yale University, New Haven, Connecticut, United States.ORCID 0009-0003-6615-1258 Gavin MaysDepartment of Biomedical Engineering, Yale University, New Haven, Connecticut, United States.ORCID 0000-0001-9727-2026 Edward P ManningPulmonary and Critical Care Medicine, Yale School of Medicine, New Haven, Connecticut, United States.ORCID 0000-0002-4430-8168 TuKiet T LamDepartment of Molecular Biophysics and Biochemistry, Yale University, New Haven, Connecticut, United States.ORCID 0000-0002-4850-3462 Jay D HumphreyDepartment of Biomedical Engineering, Yale University, New Haven, Connecticut, United States.ORCID 0000-0003-1011-2025 Dar WeissDepartment of Biomedical Engineering, Yale University, New Haven, Connecticut, United States.ORCID 0000-0001-6097-6038 Funding
TGFb-Dependent Cell-Matrix Interactions in Thoracic AortopathyP01HL169168 · NHLBI · YALE UNIVERSITY · PI Jay D. Humphrey · 2025 to 2026
$7.4MNeural Operator Learning to Predict Aneurysmal Growth and OutcomesR01HL168473 · NHLBI · YALE UNIVERSITY · PI Roland Assi, Chiara Bellini · 2023 to 2026
$2.7MELUCIDATING MECHANISMS OF THORACIC AORTOPATHY VIA COMPUTATIONAL MODELINGR01HL169147 · NHLBI · YALE UNIVERSITY · PI Jay D. Humphrey, George Tellides · 2024 to 2026
$2.0M6500 QTrap Mass Spectrometer for Yale UniversityS10OD018034 · OD · YALE UNIVERSITY · PI MANE, SHRIKANT M · 2014 to 2014
$514kAge-related Changes in the Pulmonary ArteryR03AG074063 · NIA · YALE UNIVERSITY · PI MANNING, EDWARD P · 2022 to 2023
$335kAn Ultra-Performance Liquid Chromatography System to Support Metabolomics at Yale UniversityS10OD019967 · OD · YALE UNIVERSITY · PI LAM, TUKIET T · 2015 to 2015
$135kFondation Leducq (Leducq Foundation) erADDicateFoundation for the National Institutes of Health (FNIH) AG074063Foundation for the National Institutes of Health (FNIH) HL169147Foundation for the National Institutes of Health (FNIH) HL169168Foundation for the National Institutes of Health (FNIH) S10OD02365101NHLBI NIH HHS P01 HL169168NHLBI NIH HHS R01 HL168473NHLBI NIH HHS R01 HL169147NIA NIH HHS R03 AG074063NIH HHS S10 OD018034NIH HHS S10 OD019967
6 · The paper itselfAbstract
There remains a pressing need to identify pathological mechanisms that render a thoracic aortic aneurysm susceptible to continued enlargement, dissection, or rupture, yet additional insight can be gleaned by understanding compensatory mechanisms that limit disease progression and thereby stabilize a lesion. Our biomechanical data suggest that the ascending aorta within a common mouse model of Marfan syndrome,
Indexed as
AgingAneurysm, Ascending AortaMarfan SyndromeAdipokinesAge FactorsAnimalsAorta, ThoracicDisease Models, AnimalDisease ProgressionFibrillin-1MaleMicePhenotypeProteomicsSignal TransductionTOR Serine-Threonine KinasesAdipokinesFbn1 protein, mouseFibrillin-1mTOR protein, mouseTOR Serine-Threonine KinasesTransforming Growth Factor betaaortic stiffnessfibrillin-1mTORproteometranscriptome
Identifiers
PMID42222959
PMCPMC13471068
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