ArticleBiology of sport2026
Associations of physical activity domains and delayed biological aging: assessing heterogeneity and interaction of effects.
Article in Biology of sport, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Physical activity (PA) is widely recognized for its physical and mental health benefits, but the effects of different PA domains on delayed biological aging remain inconclusive. This study aimed to investigate the specific effects of occupational PA (OPA), transportation PA (TPA), and leisure-time PA (LTPA) on delayed biological aging, as well as the heterogeneity and joint interactions of these effects. We analyzed data from 18,362 adults in NHANES 2007-2010 and 2015-2018. Biological aging was assessed using Klemera-Doubal Method Biological Age (KDM-BA), Phenotypic Age (PhenoAge), and Homeostatic Dysregulation (HD). Delayed aging was defined as biological age acceleration < 0 for KDM-BA and PhenoAge, and as values below the median for HD. Logistic regression models were applied to estimate associations of PA domains and levels with delayed aging, and joint analyses were conducted to examine combined effects. Results showed that high OPA was associated with a lower likelihood of delayed aging (KDM, OR=0.84, 95% CI=0.78-0.91; PhenoAge, OR=0.86, 95% CI=0.79-0.94), whereas high LTPA (PhenoAge, OR=1.35, 95% CI=1.22-1.49; HD, OR=1.18, 95% CI=1.09-1.29) was associated with a higher likelihood, while associations for TPA were not significant after adjustment. Joint analysis showed that combinations with high OPA were associated with lower likelihood of delayed aging, whereas those with high LTPA were associated with higher likelihood. These findings highlight the distinct roles of different PA domains and levels in delaying biological aging, providing important evidence for public health strategies to promote healthy aging.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.