ArticleJACC. Case reports2026
Severe Renal Colic as a Trigger of Coronary Thrombosis on Plaque Erosion.
Article in JACC. Case reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundAcute myocardial infarction may result from the complex interplay between transient systemic triggers and vulnerable coronary substrates. CASE SUMMARY: A 37-year-old man presented with persistent chest pain after severe renal colic. Electrocardiography demonstrated lateral ST-segment elevation with diffuse ST-segment depression. Emergency coronary angiography revealed thrombotic occlusion of the proximal left anterior descending artery. Optical coherence tomography (OCT) identified extensive thrombus overlying a mildly atherosclerotic segment without evidence of plaque rupture, consistent with probable plaque erosion. Thrombus aspiration followed by drug-coated balloon angioplasty restored TIMI flow grade 3 without stent implantation. DISCUSSION: This case illustrates a possible trigger-substrate interaction, in which severe visceral pain may act as a systemic trigger for coronary thrombosis in a patient with an erosion-prone phenotype. OCT was pivotal in defining the underlying mechanism and guiding a stent-free strategy. TAKE-HOME MESSAGES: Transient systemic stressors may act as triggers of acute coronary events. OCT enables mechanism-based, stent-free management in selected cases of plaque erosion.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.