In one paragraphArticle in mBio, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
10 authors.
Nawal Abdul-BakiDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.ORCID 0009-0005-4389-4715 Austin NegronDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.
Reimi NavarroDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.
Althea CampuzanoDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.
Matthew Mendoza BarkerDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.
Nicolas D PratherDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.
Paul L FidelCenter of Excellence in Oral and Craniofacial Biology, School of Dentistry, Louisiana State University Health Sciences Center, New Orleans, Louisiana, USA.ORCID 0000-0001-8066-5763 Thomas G ForsthuberDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.ORCID 0000-0003-0692-7125 Jieh-Juen YuDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.ORCID 0000-0001-8901-3366 Chiung-Yu HungDepartment of Molecular Microbiology and Immunology, South Texas Center for Emerging Infectious Diseases, The University of Texas at San Antonio, San Antonio, Texas, USA.ORCID 0000-0003-1091-3420 Funding
Screening Repurposing Libraries for the Identification of Drugs with Novel anti-Coccidioidal ActivityU19AI166761 · NIAID · UNIVERSITY OF TEXAS SAN ANTONIO · PI CHIUNG-YU HUNG · 2022 to 2026
$8.1MDevelopment of a multivalent vaccine against Coccidioides infectionR01AI135005 · NIAID · UNIVERSITY OF TEXAS SAN ANTONIO · PI HUNG, CHIUNG-YU · 2018 to 2022
$1.9MNIAID NIH HHS R01 AI135005NIAID NIH HHS U19 AI166761
6 · The paper itselfAbstract
Coccidioidomycosis is a pulmonary disease caused by the dimorphic fungus IMPORTANCE: Dimorphic fungi are primary pathogens that infect both immunocompetent and immunocompromised hosts. Fungal pathogens have evolved sophisticated strategies to evade innate immune responses, which have not been well studied. Recently, myeloid-derived suppressor cells (MDSCs) have been described in the pathogenesis of several fungal infections, including
Indexed as
CD4-Positive T-LymphocytesCoccidioidesCoccidioidomycosisLung Diseases, FungalMyeloid-Derived Suppressor CellsNeutrophilsAnimalsAntigens, LyDisease Models, AnimalFemaleLungMiceMice, Inbred C57BLAntigens, LyLy6G antigen, mouseCoccidioidescoccidioidomycosismyeloid-derived suppressor cellsT cell proliferationValley fever
Identifiers
PMID42206847
PMCPMC13343843
What OpenQuestion holds
Textmetadata
LicenceCC BY
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