Evidence map›Paper›PMID 42195050›Full record

ReviewGenes2026

Role of Caveolin-1 in Inflammation: Genetic Predisposition and Potential Implication for Multiple Sclerosis.

Bruk Getachew, Matthew R Miller, Harold E Landis, Robert E Miller, Yousef Tizabi

Abstract readReview
In one paragraph

Review in Genes, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Bruk GetachewDepartment of Pharmacology, Howard University College of Medicine, Washington, DC 20059, USA.ORCID 0000-0003-3523-2154
Matthew R MillerNutriGenetic Research Institute, Ephrata, PA 17522, USA.
Harold E LandisIntegrative Medicine Fellow, University of Arizona, Tucson, AZ 85724, USA.
Robert E MillerNutriGenetic Research Institute, Ephrata, PA 17522, USA.
Yousef TizabiDepartment of Pharmacology, Howard University College of Medicine, Washington, DC 20059, USA.ORCID 0000-0003-1703-1842

Funding

National Institute of Health 2 SO6 GM08016-39
6 · The paper itself

Abstract

Multiple Sclerosis (MS) is a chronic, immune-mediated disorder of the central nervous system characterized by leukocyte infiltration, inflammation, demyelination, and progressive neurodegeneration. Susceptibility to MS is influenced by genetic factors, including variants within the human leukocyte antigen (

Indexed as

Caveolin 1Genetic Predisposition to DiseaseInflammationMultiple SclerosisAnimalsHumansSignal TransductionCAV1 protein, humanCaveolin 1autophagyBBBcaveolaeCaveolin-1genetic modelHLA-DR15ICAMinflammationMSoxidative stresssystems biology

Identifiers

PMID42195050
PMCPMC13206041

What OpenQuestion holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.