Article in Human mutation, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
10 authors.
Shan LiCenter for Rare Diseases, State Key Laboratory of Complex, Severe, and Rare Diseases, Peking Union Medical College Hospital, Peking Union Medical College and Chinese Academy of Medical Sciences, Beijing, China, pumch.cn.ORCID https://orcid.org/0000-0002-0272-6193
Xiumin ChenState Key Laboratory for Complex, Severe, and Rare Diseases, Department of Medical Genetics, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and School of Basic Medicine, Peking Union Medical College, Beijing, China, pumc.edu.cn.ORCID https://orcid.org/0000-0003-2107-5071
Yixuan CaoCenter for Rare Diseases, State Key Laboratory of Complex, Severe, and Rare Diseases, Peking Union Medical College Hospital, Peking Union Medical College and Chinese Academy of Medical Sciences, Beijing, China, pumch.cn.ORCID https://orcid.org/0000-0002-6025-637X
Mingchen HanState Key Laboratory for Complex, Severe, and Rare Diseases, Department of Medical Genetics, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and School of Basic Medicine, Peking Union Medical College, Beijing, China, pumc.edu.cn.ORCID https://orcid.org/0009-0001-1661-5822
Feifei GuanNational Human Diseases Animal Model Resource Center, Institute of Laboratory Animal Sciences, Peking Union Medicine College and Chinese Academy of Medical Sciences, Beijing, China.ORCID https://orcid.org/0000-0002-1627-2452
Xiuzhi RenKey Laboratory in Science and Technology Development Project of Suzhou, Children's Hospital of Soochow University, Suzhou, China.ORCID https://orcid.org/0009-0000-3440-6285
Huan MiState Key Laboratory for Complex, Severe, and Rare Diseases, Department of Medical Genetics, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and School of Basic Medicine, Peking Union Medical College, Beijing, China, pumc.edu.cn.ORCID https://orcid.org/0009-0003-2003-5040
Tao YangState Key Laboratory for Complex, Severe, and Rare Diseases, Department of Medical Genetics, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and School of Basic Medicine, Peking Union Medical College, Beijing, China, pumc.edu.cn.ORCID https://orcid.org/0000-0001-9098-0134
Mei LiDepartment of Endocrinology, Key Laboratory of Endocrinology, National Health and Family Planning Commission, Peking Union Medical College, Chinese Academy of Medical Sciences, Beijing, China, nhfpc.gov.cn.ORCID https://orcid.org/0000-0001-7270-4902
Xiuli ZhaoCenter for Rare Diseases, State Key Laboratory of Complex, Severe, and Rare Diseases, Peking Union Medical College Hospital, Peking Union Medical College and Chinese Academy of Medical Sciences, Beijing, China, pumch.cn.ORCID https://orcid.org/0000-0001-8238-6311
Funding
No grant is acknowledged in the PubMed record.
6 · The paper itself
Abstract
Osteogenesis imperfecta (OI) is a genetically and clinically heterogeneous bone disorder, with more than 20 genes contributing to OI development. Previously, we identified
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.