ArticleCase reports in vascular medicine2026
Myocardial Infarction With Nonobstructive Coronary Arteries With Coronary Microvascular Dysfunction Associated With Systemic Sclerosis: Case Report.
Article in Case reports in vascular medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Myocardial Infarction With Nonobstructive Coronary Arteries With Coronary Microvascular Dysfunction Associated With Systemic Sclerosis: Case Report.Case reports in vascular medicine · 2026Article
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Authors and funding
16 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background: Myocardial infarction with nonobstructive coronary arteries (MINOCA) accounts for a meaningful proportion of acute myocardial infarction presentations and encompasses heterogeneous mechanisms. Coronary microvascular dysfunction (CMD) is a major cause of MINOCA and can be invasively characterized using coronary physiology indices, including the index of microvascular resistance (IMR) and coronary flow reserve (CFR). Systemic sclerosis (SSc) is associated with small-vessel vasculopathy and may predispose individuals to CMD. However, MINOCA attributable to SSc-related CMD confirmed by invasive coronary function testing and cardiac magnetic resonance (CMR) is not widely reported. Case Presentation: A 76-year-old woman with SSc and interstitial pneumonia presented with persistent chest pain. Electrocardiography revealed new T-wave inversions in leads V1-V4, and echocardiography demonstrated mid-anterior left ventricular asynergy. Troponin T was elevated (0.206 ng/mL). Emergency coronary angiography showed no obstructive epicardial stenosis, but left ventriculography confirmed regional wall-motion abnormality. CMR demonstrated base-to-mid anteroseptal dysfunction with increased native T1, T2, and extracellular volume fraction, supporting MINOCA. Transient systemic inflammation was observed (peak C-reactive protein 14.69 mg/dL; white blood cell count 24,600/ Conclusion: SSc-associated MINOCA, possibly driven by CMD, was diagnosed by comprehensive invasive coronary function testing and CMR. Serial CMR may help to document reversibility of microvascular ischemic injury and guide vasodilator therapy and follow-up.
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