Article in Science advances, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
14 authors.
Lu ZhengDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.ORCID 0009-0003-5240-6117
Libiao PanDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.ORCID 0009-0007-5303-9047
Xiaoyu FuLiangzhu Laboratory, State Key Laboratory of Brain-Machine Intelligence, Zhejiang University, Hangzhou 311121, China.ORCID 0009-0003-0572-119X
Siyu Wang 王思羽Department of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.ORCID 0009-0006-0317-3734
Yue WuDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.ORCID 0009-0004-0807-9451
Hanyang XiaoDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.
Jiachao YangDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.
Siyu Wang 王思雨Department of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.
Li YangDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.
Xiaotong WuDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.ORCID 0000-0001-8979-4780
Fada PanDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.
Hongbin YangLiangzhu Laboratory, State Key Laboratory of Brain-Machine Intelligence, Zhejiang University, Hangzhou 311121, China.ORCID 0000-0001-8574-1542
Gao ChenDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.ORCID 0000-0003-1085-0028
Hao WangDepartment of Neurosurgery of Second Affiliated Hospital and School of Brain Science and Brain Medicine, Zhejiang Key Laboratory of Research and Transformation for Major Neurosurgical Diseases, Key Laboratory for Biomedical Engineering of Education Ministry, Zhejiang University School of Medicine, Hangzhou 310058, Zhejiang, China.ORCID 0000-0003-1862-8229
Funding
No grant is acknowledged in the PubMed record.
6 · The paper itself
Abstract
The medial amygdala (MeA) processes social olfactory cues, but its precise neural mechanisms remain unclear. We identified the posteroventral MeA (MeApv) as critical for individual conspecific odor discrimination in mice. Exposure to conspecifics or their odors markedly elevates calcium signals and c-Fos expression in MeApv VGluT2-positive neurons. Optogenetic silencing of these neurons or activating Gad2-positive neurons disrupts odor-driven social behaviors, including identity recognition, odor discrimination, and sex discrimination. Social information is directly relayed from the accessory olfactory bulb (AOB) to the MeApv, and acute AOB-MeApv pathway disruption impairs social discrimination. A distinct MeApv VGluT2-positive neuron population encodes individual-specific cues, as revealed by microendoscopic calcium imaging at a single-cell resolution. Selective silencing of these neurons induces deficits in odor-guided social interactions with related conspecifics, confirming the MeApv as a central hub for social information encoding. These findings establish the MeApv's dual necessity and sufficiency in translating olfactory signals into social behavioral responses.
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
The posteroventral part of the medial amygdala nucleus glutamatergic neurons encodes conspecifics' individual identity in rodents. · full record | OpenQuestion